Abstract
It has long been recognized that certain ordinarily salutary drugs may produce an acute hemolytic anemia in some susceptible individuals. The 8-aminoquinoline antimalarial, pamaquine (plasmoquine), was such a medication. It was the investigation of the hemolytic effect of pamaquine and its derivatives which led to the recognition that hereditary red cell enzyme deficiencies could produce hemolytic disease.
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References
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© 1978 Plenum Publishing Corporation
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Beutler, E. (1978). Glucose-6-Phosphate Dehydrogenase Deficiency. In: Hemolytic Anemia in Disorders of Red Cell Metabolism. Topics in Hematology. Springer, Boston, MA. https://doi.org/10.1007/978-1-4684-2457-7_2
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