Abstract
Atherosclerosis is a multifactorial disease whose pathogenesis is still unclear. Mounting evidence, however, supports the concept that subendothelial retention of apoB100-containing lipoproteins is the initiating event in atherogenesis. Subsequently, a series of biological responses to this retained material leads to specific molecular and cellular processes that promote lesion formation.
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Received 3 July 2003; received after revision 13 August 2003; accepted 15 August 2003
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Gustafsson, M., Flood, C., Jirholt, P. et al. Retention of atherogenic lipoproteins in atherogenesis. CMLS, Cell. Mol. Life Sci. 61, 4–9 (2004). https://doi.org/10.1007/s00018-003-3262-x
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DOI: https://doi.org/10.1007/s00018-003-3262-x