Article PDF
Avoid common mistakes on your manuscript.
References
Beranek JT (2001) Pathogenesis of transverse midventricular disruption. Int J Cardiol 81:289–290
Ammann P, Fehr T, Minder EI, Gunter C, Bertel O (2001) Elevation of troponin I in sepsis and septic shock. Intensive Care Med 27:965–969 DOI 10.1007/s001340100920
Robbins SL, Cotran RS, Kumar V (1984) Pathologic basis of disease. Saunders, Philadelphia, pp 112–117
Kleinman WM, Krause SM, Hess ML (1980) Differential subendocardial perfusion and injury during the course of Gram-negative endotoxemia. Adv Shock Res 4:139–152
McGovern VJ, Tiller DJ (1980) Shock. A clinicopathologic correlation. Masson, New York, pp 17–77
Wu AHB (2001) Increased troponin in patients with sepsis and septic shock: myocardial necrosis or reversible myocardial depression? Intensive Care Med 27:959–961 DOI 10.1007/s001340100970
Bowen FW, Hattori T, Narula N, Salgo IS, Plappert T, St. John Sutton MG, Edmunds LH Jr (2001) Reappearance of myocytes in ovine infarcts produced by six hours of complete ischemia followed by reperfusion. Ann Thorac Surg 71:1845–1855
Author information
Authors and Affiliations
Rights and permissions
About this article
Cite this article
Beranek, J.T. Cardiomyocyte apoptosis contributes to the pathology of the septic shock heart. Intensive Care Med 28, 218 (2002). https://doi.org/10.1007/s00134-001-1181-6
Received:
Accepted:
Published:
Issue Date:
DOI: https://doi.org/10.1007/s00134-001-1181-6