Abstract
Maintenance of the cortical EP depends basically on the adequacy of tissue blood flow at all levels of the afferent pathway and, in particular, on cortical flow. To extend the clinical use of the EP to the monitoring of functional recovery following acute cerebrovascular lesions, such as stroke or vasospasm, it must first be established how critically the cortical EP depends on the depth of ischaemia in cortex (the degree of blood flow reduction) and on its duration. More fundamentally, the pathophysiology of the EP is also of interest, especially in relation to metabolic impairment caused by ischaemia.
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References
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© 1980 MTP Press Limited
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Branston, N.M., Symon, L. (1980). Cortical EP, blood flow and potassium changes in experimental ischaemia. In: Barber, C. (eds) Evoked Potentials. Springer, Dordrecht. https://doi.org/10.1007/978-94-011-6645-4_61
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DOI: https://doi.org/10.1007/978-94-011-6645-4_61
Publisher Name: Springer, Dordrecht
Print ISBN: 978-94-011-6647-8
Online ISBN: 978-94-011-6645-4
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