Abstract
Some years ago, for lack of better knowledge, most of us were tempted to classify shock on an etiologic basis. We referred to: (a) shock due to blood loss (hypovolemic shock); (b) cardiogenic shock (pump failure); (c) bacterial shock (bacteremia, endotoxin); (d) acute hypersensitivity reactions (anaphylactic shock); (e) neurogenic shock (barbiturate and narcotic overdose, transection of the spinal cord; (f) shock associated with obstruction to blood flow, e. g., pulmonary embolization; and (g) endocrine forms of shock (myxedema, pheochromocytoma, adrenocortical insufficiency, insulin excess).
This study was supported by USPHS Research Grants HE-00570 from the National Heart and Lung Institute, GM-16462 from the National Institute of General Medical Sciences, and HS-00238 from the National Center for Health Services and Development, HSMHA.
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References
Shubin, H., and M.H. Weil. The mechanism of shock following suicidal doses of barbiturates, narcotics, and tranquilizer drugs, with observations on the effects of treatment. Am. J. Med. 38: 857, 1965.
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Weil, M.H., and H. Shubin. Diagnosis and Treatment of Shook. Baltimore: Williams amp; Wilkins, 1967, p. 10.
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© 1972 Plenum Press, New York
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Weil, M.H., Shubin, H. (1972). Proposed Reclassification of Shock States with Special Reference to Distributive Defects. In: Hinshaw, L.B., Cox, B.G. (eds) The Fundamental Mechanisms of Shock. Advances in Experimental Medicine and Biology, vol 23. Springer, Boston, MA. https://doi.org/10.1007/978-1-4615-9014-9_3
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DOI: https://doi.org/10.1007/978-1-4615-9014-9_3
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